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SYS · ONLINEPASS · 63.0%
Open Assay
Independent Testing / Est. 2026
BATCH04·26·B
PASS63.0%
N27
PeptidesCosmeticPalmitoyl Tripeptide-5

Palmitoyl Tripeptide-5

/ Synthetic palmitoylated tripeptide; INCI 'Palmitoyl Tripeptide-5'; TGF-β mimetic cosmetic peptide
SPECULATIVEN = 0 · TESTING PENDINGMW 523.80 g·mol⁻¹

ALIAS · Syn-Coll (trade) · Palmitoyl Tripeptide-5 (INCI) · Pal-KVK

Pass rate
0
Samples
0
Suppliers
Research use onlyAny dose figures below describe what specific cited studies used, reported factually. Nothing on this page is guidance for human use.READ FIRST →

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§ A · Identity
Primary sequencePal-KVK
MW · 523.80CLASS · Synthetic palmitoylated tripeptide; INCI 'Palmitoyl Tripeptide-5'; TGF-β mimetic cosmetic peptideCATEGORY · Cosmetic

Tier 4. DSM/Pentapharm's Syn-Coll (Palmitoyl Tripeptide-5) is marketed as a TGF-β-mimetic cosmetic peptide intended to drive collagen synthesis through TGF-β-receptor pathway engagement. Supplier-authored ingredient dossiers describe in-vitro collagen-induction data; peer-reviewed independent literature on the specific INCI is limited.

§ B · Mechanism of action

The supplier-described mechanism is mimicry of thrombospondin-1's TGF-β-activating motif: the KVK tripeptide is presented as a bioactive fragment that promotes activation of latent TGF-β at the dermal level, with downstream collagen-synthesis effects in fibroblasts. Palmitoylation increases lipophilicity for topical delivery. Independent peer-reviewed mechanism characterisation in the cosmetic context is limited.

§ C · Human clinical evidence

Tier 4. Cell-culture collagen-induction data and finished-product cosmetic trials are predominantly supplier-authored. PubMed-indexed independent primary literature on Palmitoyl Tripeptide-5 in vivo on human skin is sparse.

§ F · Safety signal

No published independent safety database. Cosmetic INCI status implies regulatory acceptance for topical use. Theoretical concern about TGF-β-pathway agonism (TGF-β biology connects to fibrosis and tumour-microenvironment remodelling) is generally considered low at topical cosmetic concentrations but has not been formally characterised.

§ H · Regulatory status

Regulatory status

FDA status:
Not FDA-approved
§ I · Notable gaps and controversies

The thrombospondin-derived TGF-β-activation rationale is biologically interesting but the published evidence that topical Pal-KVK measurably activates TGF-β in human skin in vivo is industry-authored. Independent replication is limited.