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SYS · ONLINEPASS · 63.0%
Open Assay
Independent Testing / Est. 2026
BATCH04·26·B
PASS63.0%
N27
PeptidesMetabolicMaralixibat

Maralixibat

/ Selective ileal bile acid transporter (IBAT/ASBT) inhibitor (small molecule)
TIER 1 · ClinicalN = 0 · TESTING PENDINGMW 710.40 g·mol⁻¹

ALIAS · LIVMARLI (trade) · Maralixibat chloride · SHP-625 · LUM-001

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Research use onlyAny dose figures below describe what specific cited studies used, reported factually. Nothing on this page is guidance for human use.READ FIRST →

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§ A · Identity
Primary sequence— sequence not captured —
MW · 710.40CLASS · Selective ileal bile acid transporter (IBAT/ASBT) inhibitor (small molecule)CATEGORY · Metabolic

Tier 1. Maralixibat (LIVMARLI) is a non-systemic, minimally absorbed selective ileal bile acid transporter (IBAT/ASBT) inhibitor. FDA approval 2021 for cholestatic pruritus in patients with Alagille syndrome aged 3 months and older, based on the ICONIC Phase 2 trial. Indication expanded in 2024 to progressive familial intrahepatic cholestasis (PFIC) based on the EMBARK Phase 3 program.

§ B · Mechanism of action

Maralixibat is a selective small-molecule inhibitor of the apical sodium-dependent bile acid transporter (ASBT, also called IBAT), expressed on the luminal surface of terminal ileal enterocytes. Inhibition of intestinal bile acid reabsorption interrupts the enterohepatic circulation, increases faecal bile acid excretion, lowers serum bile acid concentrations, and reduces the cholestatic pruritus burden that defines Alagille syndrome and certain PFIC subtypes. The compound is minimally systemically absorbed; the pharmacology is luminal.

§ C · Human clinical evidence

Tier 1. The ICONIC Phase 2 trial (Gonzales and colleagues 2021 with subsequent open-label extension) randomised paediatric Alagille syndrome participants with cholestatic pruritus to maralixibat or placebo and reported reduced pruritus scores and serum bile acids; long-term extension data through five years (Loomes and colleagues, Hepatology 2021) supported durability of effect. The EMBARK Phase 3 trial (Karpen and colleagues, Lancet 2023) extended the evidence base into PFIC.

§ D · Primary literature
PubMed34755627Gonzales E et al.Efficacy and safety of maralixibat treatment in patients with Alagille syndrome and cholestatic pruritus (ICONIC): a randomised phase 2 study · The Lancet · human-phase-2In Alagille syndrome paediatric participants with cholestatic pruritus, maralixibat reduced itch severity and serum bile acids versus placebo; randomised withdrawal phase confirmed treatment effect on pruritus and bile acid endpoints.Limitations: Small randomised cohort; orphan indication; long-term outcomes assessed in open-label extension.2021
PubMed35507739Loomes KM et al.Maralixibat for the treatment of PFIC: long-term, IBAT inhibition in an open-label, Phase 2 study · Hepatology Communications · human-phase-2Long-term open-label maralixibat in PFIC participants demonstrated sustained reductions in pruritus and serum bile acids over multi-year follow-up, supporting durability of the IBAT-inhibitor pharmacology.Limitations: Open-label, no concurrent control; selection for tolerators.2022
§ F · Safety signal

Common adverse events are gastrointestinal (diarrhoea, abdominal pain) related to the increased luminal bile acid load and altered bile acid pool composition. Liver enzyme elevations and fat-soluble vitamin malabsorption secondary to interrupted bile acid recycling have been reported and require monitoring. Class concerns about long-term effects on enterohepatic bile acid biology in growing children remain under post-marketing surveillance.

§ H · Regulatory status

Regulatory status

FDA status:
FDA-approved
Compounding:
Not eligible for compounding (approved, not in shortage)
§ I · Notable gaps and controversies

Maralixibat is one of two IBAT inhibitors approved in this orphan indication space (the other is odevixibat, approved earlier for PFIC). Comparative head-to-head trials between the two have not been performed; cross-trial comparisons are confounded by trial design and population differences. Vendor research-chemical use is not relevant — this is a prescription orphan drug with regulated paediatric administration.