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SYS · ONLINEPASS · 63.0%
Open Assay
Independent Testing / Est. 2026
BATCH04·26·B
PASS63.0%
N27
PeptidesMetabolicGlucagon (rescue)

Glucagon (rescue)

/ Recombinant 29-residue glucagon peptide; native glucagon receptor agonist
TIER 1 · ClinicalN = 0 · TESTING PENDINGMW 3482.80 g·mol⁻¹

ALIAS · GlucaGen (trade) · Glucagon Emergency Kit · Recombinant glucagon · Baqsimi (intranasal) · Gvoke (auto-injector)

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Research use onlyAny dose figures below describe what specific cited studies used, reported factually. Nothing on this page is guidance for human use.READ FIRST →

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§ A · Identity
Primary sequenceHSQGTFTSDYSKYLDSRRAQDFVQWLMNT
MW · 3482.80CLASS · Recombinant 29-residue glucagon peptide; native glucagon receptor agonistCATEGORY · Metabolic

Tier 1. Recombinant 29-residue glucagon is FDA-approved across multiple formulations for severe hypoglycemia rescue: the legacy lyophilised reconstitute-and-inject Glucagon Emergency Kit and GlucaGen, the ready-to-use Gvoke autoinjector and pre-filled syringe (2019), and the intranasal Baqsimi powder (2019). Pivotal non-inferiority trials versus reconstituted glucagon supported the newer formulations.

§ B · Mechanism of action

Glucagon is the 29-residue peptide hormone secreted by pancreatic alpha cells in response to hypoglycemia. Receptor binding at the hepatic glucagon receptor activates adenylate cyclase via Gs-alpha, raising intracellular cAMP and driving glycogenolysis and gluconeogenesis. The resulting rapid release of hepatic glucose into the systemic circulation is the molecular basis for severe-hypoglycemia rescue. Recombinant glucagon is sequence-identical to the endogenous human peptide.

§ C · Human clinical evidence

Tier 1. The Baqsimi intranasal powder pivotal program (Rickels 2016 Diabetes Care, Pieber 2018 Diabetes Care) demonstrated non-inferiority to intramuscular reconstituted glucagon for plasma glucose recovery in adults and adolescents with type 1 diabetes during insulin-induced hypoglycemia. The Gvoke ready-to-use autoinjector program demonstrated comparable efficacy with substantially reduced administration complexity versus the legacy reconstitute-and-inject kit. The legacy product itself has decades of clinical-use evidence in severe hypoglycemia.

§ D · Primary literature
PubMed26681725Rickels MR et al.Intranasal glucagon for treatment of insulin-induced hypoglycemia in adults with type 1 diabetes: a randomized crossover noninferiority study · Diabetes Care · human-phase-3-rctIntranasal glucagon 3 mg was non-inferior to intramuscular glucagon 1 mg for plasma glucose recovery during insulin-induced hypoglycemia in adults with type 1 diabetes; treatment success was 98.7% intranasal versus 100% intramuscular.Limitations: Crossover design with insulin-induced hypoglycemia in clinical setting, not real-world severe-hypoglycemia events; small sample size.2016
§ F · Safety signal

Common adverse events across formulations include nausea, vomiting, headache, and injection-site or nasal-administration-site reactions. Hyperglycemic rebound after rescue is expected. Glucagon is contraindicated in pheochromocytoma (catecholamine release) and insulinoma (paradoxical insulin release). Effectiveness depends on hepatic glycogen stores; in severe malnutrition, prolonged fasting, or chronic alcoholism, response may be attenuated.

§ H · Regulatory status

Regulatory status

FDA status:
FDA-approved
Compounding:
Not eligible for compounding (approved, not in shortage)
§ I · Notable gaps and controversies

The transition from reconstitute-and-inject to ready-to-use formulations addressed a long-standing usability problem in severe-hypoglycemia rescue; the older kit had documented administration-error rates in panic situations. The newer formulations have not changed pharmacology — only delivery. Bihormonal closed-loop systems and dasiglucagon (a stable analog) are adjacent developments that go beyond rescue dosing.