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SYS · ONLINEPASS · 63.0%
Open Assay
Independent Testing / Est. 2026
BATCH04·26·B
PASS63.0%
N27
PeptidesCognitiveAdrafinil

Adrafinil

/ Pro-drug of modafinil (small molecule, hydroxyamide of modafinil)
TIER 2 · TranslationalN = 0 · TESTING PENDINGMW 289.35 g·mol⁻¹

ALIAS · Olmifon (trade — formerly EU)

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Research use onlyAny dose figures below describe what specific cited studies used, reported factually. Nothing on this page is guidance for human use.READ FIRST →

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§ A · Identity
Primary sequence— sequence not captured —
MW · 289.35CLASS · Pro-drug of modafinil (small molecule, hydroxyamide of modafinil)CATEGORY · Cognitive

Tier 2. Was approved in France and several EU countries as Olmifon for narcolepsy and age-related vigilance disorders from the 1980s. Voluntarily withdrawn by Cephalon in 2011 — a business decision linked to focus on its active metabolite modafinil rather than a safety-driven action. Not approved in the US. Sold in the US through nootropic vendor channels; no longer available through any approved pharmaceutical channel in any major market.

§ B · Mechanism of action

Adrafinil is a hydroxyamide pro-drug that is hepatically metabolised to modafinil (and to the inactive modafinil-acid metabolite). The wakefulness-promoting pharmacology is therefore inherited from modafinil, which has weak dopamine-reuptake inhibition and effects on histamine, orexin, and noradrenaline systems. Because conversion is incomplete and slower than direct modafinil dosing, adrafinil produces a delayed onset and lower modafinil exposure per milligram than modafinil itself. The hepatic conversion is the basis for adrafinil's higher liver-enzyme-elevation profile relative to modafinil.

§ C · Human clinical evidence

Tier 2. The published human-evidence base centres on aged-population vigilance and cognitive studies in Europe (1980s–1990s, principally French-language literature) and on the canine cognitive-aging model (Milgram, Siwak, and colleagues). The canine work is methodologically rigorous within its species and provides the most cited preclinical pharmacology, but does not substitute for contemporary human RCT data; the French elderly-vigilance literature is mostly outside indexed English-language journals.

§ D · Primary literature
PubMed10880682Milgram NW et al.Oral administration of adrafinil improves discrimination learning in aged beagle dogs · Pharmacology Biochemistry and Behavior · other-animalOral adrafinil improved performance on a two-choice visual-discrimination learning task in aged beagles relative to baseline.Limitations: Canine model; dose-finding study; behavioural endpoint specific to the species.2000
PubMed10880681Siwak CT et al.Behavioral activating effects of adrafinil in aged canines · Pharmacology Biochemistry and Behavior · other-animalAdrafinil produced dose-dependent behavioural activation (increased locomotor activity, decreased sleep) in aged beagles.Limitations: Behavioural-activation endpoint in canines; relevance to human vigilance is by inference.2000
PubMed10880681Siwak CT et al.Behavioral activating effects of adrafinil in aged canines · Pharmacology Biochemistry and Behavior · other-animalAdrafinil produced dose-dependent behavioural activation (increased locomotor activity, decreased sleep) in aged beagles.Limitations: Behavioural-activation endpoint in canines; relevance to human vigilance is by inference.2000
PubMed15795058Studzinski CM et al.The canine model of human cognitive aging and dementia: pharmacological validity of the model for assessment of human cognitive-enhancing drugs · Progress in Neuro-Psychopharmacology & Biological Psychiatry · reviewMethodological review of the canine cognitive-aging model and its pharmacological validity for human cognitive-enhancing drug evaluation; adrafinil is one of the agents discussed.Limitations: Narrative methodological review; not original adrafinil data.2005
§ F · Safety signal

Class concerns are dominated by hepatic enzyme elevation — the asymmetric carbon and the amide hydrolysis chemistry mean adrafinil produces measurable transaminase elevation more frequently than modafinil at equivalent wakefulness exposure. Stevens-Johnson syndrome and DRESS reactions documented for modafinil are read-across concerns. No comprehensive contemporary human safety database is available because Olmifon was withdrawn before modern post-marketing pharmacovigilance applied to nootropic use.

§ H · Regulatory status

Regulatory status

FDA status:
Not FDA-approved
§ I · Notable gaps and controversies

Olmifon withdrawal was a Cephalon business decision favouring modafinil rather than a safety action — but the practical effect is that adrafinil is no longer available through any approved pharmaceutical channel anywhere. Vendor-sold adrafinil in the US is not pharmaceutical-grade material and is purchased through nootropic-supplement channels that do not have the identity, purity, or hepatic-monitoring infrastructure of the original Olmifon prescription pathway. Subjects choosing modafinil directly avoid the hepatic-conversion step entirely.